Familial hypertrophic cardiomyopathy-related cardiac troponin C mutation L29Q affects Ca binding and myofilament contractility

نویسندگان

  • Bo Liang
  • Franca Chung
  • Yang Qu
  • Dmitri Pavlov
  • Todd E. Gillis
  • Svetlana B. Tikunova
  • Jonathan P. Davis
  • Glen F. Tibbits
چکیده

Bo Liang, Franca Chung, Yang Qu, Dmitri Pavlov, Todd E. Gillis, Svetlana B. Tikunova, Jonathan P. Davis, and Glen F. Tibbits Cardiac Membrane Research Laboratory, Kinesiology, Simon Fraser University, Burnaby; Cardiovascular Sciences, Child & Family Research Institute, Vancouver, British Columbia; Integrative Biology, University of Guelph, Guelph, Ontario, Canada; and Department of Physiology and Cell Biology, The Ohio State University, Columbus, Ohio

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Familial hypertrophic cardiomyopathy-related cardiac troponin C mutation L29Q affects Ca2+ binding and myofilament contractility.

The cardiac troponin C (cTnC) mutation, L29Q, has been found in a patient with familial hypertrophic cardiomyopathy. We previously showed that L29, together with neighboring residues, Asp2, Val28, and Gly30, plays an important role in determining the Ca(2+) affinity of site II, the regulatory site of mammalian cardiac troponin C (McTnC). Here we report on the Ca(2+) binding characteristics of L...

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Structural kinetics of cardiac troponin C mutants linked to familial hypertrophic and dilated cardiomyopathy in troponin complexes.

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The structural and functional effects of the familial hypertrophic cardiomyopathy-linked cardiac troponin C mutation, L29Q

Familial hypertrophic cardiomyopathy (FHC) is characterized by severe abnormal cardiac muscle growth. The traditional view of disease progression in FHC is that an increase in the Ca(2+)-sensitivity of cardiac muscle contraction ultimately leads to pathogenic myocardial remodeling, though recent studies suggest this may be an oversimplification. For example, FHC may be developed through altered...

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تاریخ انتشار 2008